X-linked Hypophosphataemic Rickets in Australian Children: Prevalence and Burden of Disease
Aim
Methods
Case definition, child <18 years with XLH: Rickets during childhood AND Pathogenic mutation in the PHEX gene OR iFGF23 levels above the limits of the local laboratory range OR Family history that supports X-linked inheritance.
The study was conducted with the Australian Paediatric Surveillance Unit (APSU). In June and July 2020, 1434 paediatricians were asked if they had seen a child with XLH. Paediatricians completed a case report form for each child into a REDCap database or sent to APSU via email.
Results:
47 paediatricians reported 1 or more cases of XLH. After exclusion of duplicates (10) and errors (5) there were 74 confirmed cases. The minimum national prevalence was 1.31 per 100,000 children under 18 years (CI 1.02 – 1.64). Current medial age 11.0 years (1-18), medial age at diagnosis 2.0 years (0.1-17), 59% female and 67% family history of XLH. PHEX gene testing in 49 children (65%) of which 47 had a pathogenic mutation. Males were more likely to have bone and joint pain, kyphosis, tooth abscess and cransiosynostosis (p<0.05).
33 (45%) were managed with phosphate and calcitriol and 41 (55%) with Burosumab, which was associated with a higher serum phosphate level. Complications of phosphate and calcitriol therapy included nephrocalcinosis (32%), hyperparathyroidism (18%) and renal failure (1%).
Conclusion:
Prevalence of XLH in Australia is similar to other reported studies. XLH is associated with a significant burden of disease and males appeared more severely affected. Diagnosis is often delayed. Burosumab normalised serum phosphate in clinical use.